Search results for "TOLL-LIKE RECEPTORS"

showing 10 items of 70 documents

Linking species habitat and past palaeoclimatic events to evolution of the teleost innate immune system

2017

Host-intrinsic factors as well as environmental changes are known to be strong evolutionary drivers defining the genetic foundation of immunity. Using a novel set of teleost genomes and a time-calibrated phylogeny, we here investigate the family of Toll-like receptor ( TLR ) genes and address the underlying evolutionary processes shaping the diversity of the first-line defence. Our findings reveal remarkable flexibility within the evolutionary design of teleost innate immunity characterized by prominent TLR gene losses and expansions. In the order of Gadiformes, expansions correlate with the loss of major histocompatibility complex class II ( MHCII ) and diversifying selection analyses sup…

0106 biological sciences0301 basic medicine1001198Evolutionpast climatic changeLineage (evolution)ClimateGenes MHC Class II199010603 evolutionary biology01 natural sciencesGenomeGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesImmune systemPhylogeneticsAnimalsGeneAtlantic Oceaninnate immunityEcosystemPhylogenyGeneral Environmental ScienceInnate immune systemadaptive evolutionGeneral Immunology and MicrobiologybiologyEcologyGadiformesToll-Like ReceptorsFishes70General Medicinegene lossbiology.organism_classificationBiological EvolutionImmunity InnateEvolvability030104 developmental biologygene expansionEvolutionary biologyImmune SystemGeneral Agricultural and Biological SciencesResearch Article
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Triggering of Toll-like Receptors in Old Individuals. Relevance for Vaccination

2019

Aging is characterized by a general decline in a range of physiological functions, with a consequent increase in the risk of developing a variety of chronic diseases and geriatric syndromes. Additionally, increasing age is accompanied by a progressive decline in both innate and acquired immune system, referred to as immunosenescence. This impaired ability to mount an efficient immune response after exposure to microorganisms or vaccines represents a major challenge in acquiring protection against pathogens in aging. Therefore, there is still a great need for vaccines that are tailored to optimally stimulate the aged immune system, thus promoting more successful aging. Various strategies ca…

0301 basic medicineAgingCellular immunityImmunosenescencemedicine.medical_treatmentDendritic cells03 medical and health sciences0302 clinical medicineImmune systemAdjuvants ImmunologicImmunityTLRDrug DiscoverymedicineHumansAgedAged 80 and overPharmacologyImmunity CellularInnate immune systembusiness.industryToll-Like ReceptorsVaccinationImmunosenescenceAcquired immune systemVaccination030104 developmental biologyImmunologyCytokinesbusinessAdjuvant030215 immunologyCurrent Pharmaceutical Design
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Amylase–Trypsin Inhibitors in Wheat and Other Cereals as Potential Activators of the Effects of Nonceliac Gluten Sensitivity

2018

Nonceliac gluten sensitivity (NCGS) is a gluten-related gastrointestinal disorder distinct from celiac disease (CD) and gluten allergy that is not easy to diagnose due to the lack of biomarkers. It is characterized by intestinal symptoms and extraintestinal manifestations with the consumption of gluten-containing foods. In contrast to CD, NCGS patients do not present a genetic predisposition or intestinal villi atrophy. Recent studies question the proinflammatory triggering activity of α-gliadin fraction contained in wheat, since it has been demonstrated that the amylase-trypsin inhibitors (ATIs) exert a strong activating effect on the innate immune response. We aimed to analyze the role of…

0301 basic medicineAllergyGlutensMedicine (miscellaneous)DiseaseFood Intolerancedigestive systemProinflammatory cytokine03 medical and health sciences0302 clinical medicineGenetic predispositionAnimalsHumansMedicineAmylaseEnzyme InhibitorsIntestinal MucosaImmunity MucosalTriticumPlant Proteinschemistry.chemical_classificationNutrition and DieteticsInnate immune systembiologybusiness.industrySecaleToll-Like Receptorsnutritional and metabolic diseasesHordeummedicine.diseaseGlutenImmunity Innatedigestive system diseases030104 developmental biologyGastrointestinal disorderchemistryImmunologybiology.protein030211 gastroenterology & hepatologyalpha-AmylasesEdible GrainTrypsin InhibitorsbusinessJournal of Medicinal Food
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TlR expression profile of human gingival margin-derived stem progenitor cells

2015

Background Gingival margin-derived stem/progenitor cells (G-MSCs) show remarkable periodontal regenerative potential in vivo. During regeneration, G-MSCs may interact with their inflammatory environment via toll-like-receptors (TLRs). The present study aimed to depict the G-MSCs TLRs expression profile. Material and Methods Cells were isolated from free gingival margins, STRO-1-immunomagnetically sorted and seeded to obtain single colony forming units (CFUs). G-MSCs were characterized for CD14, CD34, CD45, CD73, CD90, CD105, CD146 and STRO-1 expression, and for multilineage differentiation potential. Following G-MSCs’ incubation in basic or inflammatory medium (IL-1β, IFN-γ, IFN-α, TNF-α) a…

0301 basic medicineCD14GingivaCD34OdontologíaBiology03 medical and health sciencesHumansCD90Progenitor cellGeneral DentistryCells CulturedColony-forming unitOral Medicine and PathologyStem CellsResearchRegeneration (biology)Toll-Like Receptors:CIENCIAS MÉDICAS [UNESCO]Ciencias de la saludCell biology030104 developmental biologyOtorhinolaryngologyUNESCO::CIENCIAS MÉDICASImmunologyCD146SurgeryStem cellMedicina Oral Patología Oral y Cirugia Bucal
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Extracellular histones activate autophagy and apoptosis via mTOR signaling in human endothelial cells.

2018

Circulating histones have been proposed as targets for therapy in sepsis and hyperinflammatory symptoms. However, the proposed strategies have failed in clinical trials. Although different mechanisms for histone-related cytotoxicity are being explored, those mediated by circulating histones are not fully understood. Extracellular histones induce endothelial cell death, thereby contributing to the pathogenesis of complex diseases such as sepsis and septic shock. Therefore, the comprehension of cellular responses triggered by histones is capital to design effective therapeutic strategies. Here we report how extracellular histones induce autophagy and apoptosis in a dose-dependent manner in cu…

0301 basic medicineCell SurvivalEndothelial cellsFisiologiaApoptosisAMP-Activated Protein KinasesHistones03 medical and health sciencesExtracellularAutophagyHuman Umbilical Vein Endothelial CellsAutophagy-Related Protein-1 HomologHumansMolecular BiologyProtein kinase BPI3K/AKT/mTOR pathwaybiologyDose-Response Relationship DrugChemistryTOR Serine-Threonine KinasesAutophagyIntracellular Signaling Peptides and ProteinsAMPKNuclear ProteinsCirculating histonesCell biologyToll-like receptorsEndothelial stem cell030104 developmental biologyHistoneApoptosisbiology.proteinMolecular MedicineProto-Oncogene Proteins c-aktSignal TransductionBiochimica et biophysica acta. Molecular basis of disease
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Toll-like receptors in neuroinflammation, neurodegeneration, and alcohol-induced brain damage

2021

Toll-like receptors (TLRs) or pattern recognition receptors respond to pathogen-associated molecular patterns (PAMPs) or internal damage-associated molecular patterns (DAMPs). TLRs are integral membrane proteins with both extracellular leucine-rich and cytoplasmic domains that initiate downstream signaling through kinases by activating transcription factors like AP-1 and NF-κB, which lead to the release of various inflammatory cytokines and immune modulators. In the central nervous system, different TLRs are expressed mainly in microglia and astroglial cells, although some TLRs are also expressed in oligodendroglia and neurons. Activation of TLRs triggers signaling cascades by the host as a…

0301 basic medicineClinical BiochemistryActivating transcription factorGene ExpressionBiologyExosomesBiochemistryProinflammatory cytokine03 medical and health sciences0302 clinical medicineImmune systemGeneticsmedicineAnimalsHumansReceptorMolecular BiologyNeuroinflammationMicrogliaToll-Like ReceptorsNeurodegenerationPattern recognition receptorBrainNeurodegenerative DiseasesCell Biologymedicine.diseaseImmunity InnateCell biologyAlcoholismMicroRNAs030104 developmental biologymedicine.anatomical_structure030220 oncology & carcinogenesisNeuroinflammatory Diseases
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Gut microbiota imbalance and colorectal cancer

2016

International audience; The gut microbiota acts as a real organ. The symbiotic interactions between resident micro-organisms and the digestive tract highly contribute to maintain the gut homeostasis. However, alterations to the microbiome caused by environmental changes (e.g., infection, diet and/or lifestyle) can disturb this symbiotic relationship and promote disease, such as inflammatory bowel diseases and cancer. Colorectal cancer is a complex association of tumoral cells, non-neoplastic cells and a large amount of micro-organisms, and the involvement of the microbiota in colorectal carcinogenesis is becoming increasingly clear. Indeed, many changes in the bacterial composition of the g…

0301 basic medicineColorectal cancer[SDV]Life Sciences [q-bio]enterotoxigenic bacteroides-fragilisGut floraCyclomodulin[ SDV.CAN ] Life Sciences [q-bio]/CancerTopic Highlightstreptococcus-gallolyticus infectionbiologyGastrointestinal MicrobiomeGastroenterologyGeneral Medicinecytolethal-distending toxin3. Good healthlactobacillus-acidophilus deficientIntestinesCell Transformation NeoplasticHost-Pathogen InteractionsInflammation MediatorsColorectal NeoplasmsVirulence Factorspolymerase-chain-reaction[SDV.CAN]Life Sciences [q-bio]/CancerGut microbiotaoxidative dna-damageMicrobiologyescherichia-coli strains03 medical and health scienceshelicobacter-pylori infectionmedicineAnimalsHumansMicrobiomeBacteria[ SDV ] Life Sciences [q-bio]inflammatory-bowel-diseaseCancerHelicobacter pyloribiology.organism_classificationmedicine.diseaseStreptococcus bovisColorectal cancerGastrointestinal MicrobiomeHépatologie et Gastroentérologie030104 developmental biologytoll-like receptorsOxidative stressImmunologyHépatology and GastroenterologyDysbiosiscolorectal cancer;gut microbiota;dysbiosis;cyclomodulin;oxidative;stress;enterotoxigenic bacteroides-fragilis;oxidative dna-damage;cytolethal-distending toxin;inflammatory-bowel-disease;streptococcus-gallolyticus infection;lactobacillus-acidophilus;deficient;helicobacter-pylori infection;polymerase-chain-reaction;escherichia-coli strains;toll-like receptorsDysbiosisDNA Damage
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Diabetic microangiopathy: Pathogenetic insights and novel therapeutic approaches.

2017

Diabetic microangiopathy, including retinopathy, is characterized by abnormal growth and leakage of small blood vessels, resulting in local edema and functional impairment of the depending tissues. Mechanisms leading to the impairment of microcirculation in diabetes are multiple and still largely unclear. However, a dysregulated vascular regeneration appears to play a key role. In addition, oxidative and hyperosmolar stress, as well as the activation of inflammatory pathways triggered by advanced glycation end-products and toll-like receptors, have been recognized as key underlying events. Here, we review recent knowledge on cellular and molecular pathways of microvascular disease in diabet…

0301 basic medicineGlycation End Products AdvancedPhysiologyDiabetes retinopathyGlycation End ProductsDiseaseFibroblast growth factorHMGB1DiabeteMicrocirculationCapillary Permeability03 medical and health sciencesGlycationDiabetes mellitusmedicineSettore MED/05 - Patologia ClinicaAnimalsHumansCellular and molecular pathways; Diabetes; Diabetes retinopathy; Microangiopathy; Physiology; Molecular Medicine; PharmacologyNeovascularizationPharmacologyPathologicbiologyNeovascularization Pathologicbusiness.industryMicrocirculationMicroangiopathyDiabetesToll-Like Receptorsmedicine.diseasePrognosisCellular and molecular pathways; Diabetes; Diabetes retinopathy; Microangiopathy; Animals; Capillary Permeability; Diabetic Angiopathies; Glycation End Products Advanced; Humans; Inflammation Mediators; Microcirculation; Microvessels; Neovascularization Pathologic; Oxidative Stress; Prognosis; Signal Transduction; Toll-Like ReceptorsOxidative Stress030104 developmental biologyCellular and molecular pathwaysMicroangiopathyImmunologyMicrovesselsbiology.proteinMolecular MedicineAdvancedCellular and molecular pathwayInflammation MediatorsbusinessDiabetic AngiopathiesRetinopathySignal TransductionVascular pharmacology
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Adipocytes as a Link Between Gut Microbiota-Derived Flagellin and Hepatocyte Fat Accumulation

2016

While the role of both elevated levels of circulating bacterial cell wall components and adipose tissue in hepatic fat accumulation has been recognized, it has not been considered that the bacterial components-recognizing adipose tissue receptors contribute to the hepatic fat content. In this study we found that the expression of adipose tissue bacterial flagellin (FLG)-recognizing Toll-like receptor (TLR) 5 associated with liver fat content (r = 0.699, p = 0.003) and insulin sensitivity (r = -0.529, p = 0.016) in humans (n = 23). No such associations were found for lipopolysaccharides (LPS)-recognizing TLR4. To study the underlying molecular mechanisms of these associations, human HepG2 he…

0301 basic medicineGlycerollcsh:MedicineAdipose tissueWhite adipose tissueflagellinBiochemistryImmune ReceptorsFatsEndocrinologyAnimal CellsAdipocytesMedicine and Health SciencesInsulinlcsh:ScienceToll-like ReceptorsConnective Tissue CellsMultidisciplinaryImmune System ProteinsbiologyLiver DiseasesFatty liverin kaltaiset reseptorit [toll]Lipidsadipose tissuePhysical sciencesChemistryMitochondrial respiratory chainAdipose TissueConnective Tissuebacterial componentsCellular TypesAnatomyinsuline sensitivityResearch ArticleSignal Transductionmedicine.medical_specialtyadipocytesImmunologyMonomers (Chemistry)Gastroenterology and Hepatologyta311103 medical and health sciencesInsulin resistanceInternal medicinemedicinePolymer chemistryDiabetic Endocrinologylcsh:Rta1183ta1182Biology and Life SciencesProteinsCell Biologyliver fatmedicine.diseasehepatic fatfat accumulationHormonesIRS1Fatty LiverInsulin receptor030104 developmental biologyEndocrinologyBiological TissueTLR5biology.proteinlcsh:QPLoS ONE
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Screening of herbal extracts for TLR2- and TLR4-dependent anti-inflammatory effects.

2018

Herbal extracts represent an ample source of natural compounds, with potential to be used in improving human health. There is a growing interest in using natural extracts as possible new treatment strategies for inflammatory diseases. We therefore aimed at identifying herbal extracts that affect inflammatory signaling pathways through toll-like receptors (TLRs), TLR2 and TLR4. Ninety-nine ethanolic extracts were screened in THP-1 monocytes and HeLa-TLR4 transfected reporter cells for their effects on stimulated TLR2 and TLR4 signaling pathways. The 28 identified anti-inflammatory extracts were tested in comparative assays of stimulated HEK-TLR2 and HEK-TLR4 transfected reporter cells to dif…

0301 basic medicineLeavesHumulus lupulusTHP-1 CellsPhysiologymedicine.medical_treatmentAnti-Inflammatory AgentsDrug Evaluation Preclinicallcsh:MedicinePlant SciencePharmacologyPlant RootsImmune ReceptorsBiochemistryMonocytesWhite Blood CellsCell SignalingAnimal CellsImmune PhysiologyMedicine and Health SciencesMembrane Receptor Signalinglcsh:ScienceToll-like ReceptorsFlowering PlantsInnate Immune SystemMultidisciplinaryImmune System ProteinsbiologyOrganic CompoundsPlant AnatomyEukaryotaPlantsImmune Receptor SignalingChemistryCytokinevisual_artPhysical Sciencesvisual_art.visual_art_mediumPlant BarkCytokinesBarkSignal transductionCellular TypesResearch ArticleSignal Transductionmedicine.drug_classImmune CellsImmunologyTransfectionAnti-inflammatory03 medical and health sciencesmedicineHumansBlood CellsEthanolPlant ExtractsMacrophagesCinnamomum verumlcsh:ROrganic ChemistryOrganismsChemical CompoundsBiology and Life SciencesProteinsCell BiologyMolecular Developmentbiology.organism_classificationToll-Like Receptor 2Plant LeavesToll-Like Receptor 4TLR2030104 developmental biologyHEK293 CellsGene Expression RegulationCell cultureAlcoholsImmune Systemlcsh:QHeLa CellsDevelopmental BiologyPloS one
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